VIRAL HEPATITIS REFERS TO INFECTION OF THE LIVER CAUSED
BY A GROUP OF VIRUSES. ALL OF THESE VIRUSES PRODUCE SIMILAR PATTERNS OF CLINICAL AND MORPHOLOGIC ACUTE HEPATITIS BUT VARY
IN THEIR POTENTIAL TO INDUCE CHRONIC OR FULMINANT DISEASE OR CARRIER STATE.
INFECTIOUS HEPATITIS – HAV & HEV CAUSES A BENIGN
AND SELF LIMITED DISEASE. COINFECTION BY HDV & HBV RARELY DEVELOPS CHRONIC HEPATITIS. ACUTE COINFECTION BY HDV & HBV
VARY IN MILD TO FULMINANAT HEPATITIS. WHEREAS, PERSISTENT INFECTION AND CHRONIC HEPATITIS ARE THE HALLMARKS OF HBV & HCV
VIRUSES. INSTANCES OF SEVERE AND RAPIDLY PROGRESSIVE CHRONIC HBV & HCV HEPATITIS ARE BEING RECOGNIZED WITH INCREASING
FREQUENCY IN PATIENTS WITH HIV INFECTION.
WHERE HBV IS DNA - VIRUS, THE HCV & HIV ARE RNA
–VIRUSES. IN HBV INFECTION IMMUNOLOGICALLY MEDIATED HEPATOCYTES NECROSIS
BY SENSITIZED CYTOTOXIC T-CELLS, ATTRIBUTE TO CELLULAR EXPRESSION OF VIRAL ANTIGENS. WITH INTERANCE OF HBV-DNA INTO THE HOST
GENOME VIRAL REPLICATION CEASES, INFECTIVITY ENDS AND ACTIVE LIVER DAMAGE SUBSIDES. THE PATHOGENESIS OF THE HCV & HIV
INFECTIONS IS ALSO IMMUNOLOGICALLY MEDIATED LIVER DAMAGE. HOWEVER, HIV IS CHARACTERIZED BY PROFOUND SUPPRESSION OF T-CELL
MEDIATED IMMUNITY. WHEREIN, IN ADDITION TO T –CELL DEPLETION THERE ARE ALSO QUALITATIVE DEFECTS IN T – CELL FUNCTION
WITH A SELECTIVE LOSS OF T – CELL MEMORY. B- CELL IMMUNE ACTIVATION IN HIV – PATIENTS IS UNABLE TO MOUNT APPRECIABLE
ANTIBOBY RESPONSES TO NEW ANTIGENS, LEADING TO GREATER SEVERITY OF THE VIRAL HEPATITIS. FATAL CONDITION IS SUPPORTED BY OPPORTUNISTIC
INFECTIONS, SECONDARY NEOPLASMS, NEUROLOGIC AND METABOLIC DISORDERS. CLINICO – SYMPTOMATIC AND PATHOMECHANIC MEDICAL
TREATMENTS ENSURE LITTLE HELP, AND DEATH IS SURE.
ALL ATTEMPTS ARE MADE TO PROVIDE AUTOPATHY TREATMENTS
TO PATIENTS BEFORE THE ‘FULL BLOWN’ HBV / HCV – HIV STAGE OF THE DISEASE HAS REACHED. REGIROUS ‘TREATMENT
– ASSISTS’ IS CONTINUED. AAA – THERAPY IS STARTED AS EARLY AS POSSIBLE. BLOOD TRANSFUSION AND INTERFERON
THERAPY IS AVOIDED. HYDROCORTISONE [EFFCORLIN], ANTIBIOTICS [ROXITHROMYCIN], ANTIOXIDANT [FREE CAD] CALCIUM GLUCONATE WITH
VIT-C & B-12 [CALCIUM SANDOZ] INJECTIONS, WATER-MINERALS-GLUCOSE SUBSTITUTIONS AND VITAMIN-K INJECTIONS ARE ADDED. GRADUALLY,
THE ‘TREATMENT – ASSISTS’ IS DISCONTINUED. WITHIN THREE TO SIX MONTHS AFTER AAA - THERAPY ALONG WITH ABOVE
NOTED MEDICAMENTOSIS PATIENTS SHOW DRASTIC IMPROVEMENT. ANTIOXIDANT [ BD ], CALCIUM SANDOZ INJECTIONS [ I / M , WEEKLY ] PERITOL
/ PERICORT [ OD ] AND ANY SYMPTOMATIC TREATMENT [ IF NEEDED ] ARE CONTINUED. WITHIN NINE TO ELEVEN MONTHS AFTER THE FIRST
COURSE, THE SECOND COURSE OF AAA – THERAPY IS REINSTITUTED. PATIENTS ARE ADVISED TO CONTINUE TAKING HOT WATER –
LEMON DRINKS, THREE – FOUR TIMES A DAY, TULSI [INDIAN TERMINOLOGY] – FATT FREE CURD, AND BOILED ‘ APPLE
– GLUCOSE ‘ AS MAJOR PORTION OF THE DAILY MEAL.
MINIMUM AT THREE MONTHS INTERVALS MASTER CHECK-UP [PARAMEDICAL
INVESTIGATIONS] AND SPECIFIC AUTOPATHY TESTS ARE CERTAINLY CONDUCTED. MAJORITY [> 75%] OF THE PATIENTS RECOVERS. VERY FEW
PATIENTS NEED THIRD COURSE OF THE AAA – AUTOPATHY TREATMENT. RELAPSE OF THE HEPATITIS OF HIV / HBV – HCV HAS NOT
YET BEEN SEEN.