CANCER IS THE SECOND LEADING CAUSE OF DEATH AFTER CARDIOVASCULAR DISEASES, EVEN MORE AGONIZING THAN THE MORTALITY RATE IN CANCER IS THE EMOTIONAL & PHYSICAL SUFFERING.

CANCER / NEOPLASIA IS A MASS OF TISSUES FORMED AS A RESULT OF ABNORMAL EXCESSIVE, UNCOORDINATED, AUTONOMOUS, AND PURPOSELESS PROLIFERATION OF CELLS. IT COULD BE BENIGN OR MALIGNANT. MALIGNANT / CANCER NEOPLASIA PROLIFERATE RAPIDLY, SPREAD THROUGHOUT THE BODY AND EVENTUALLY CAUSE DEATH OF THE HOST.

THE NEOPLASTIC CELL IS CHARACTERIZED BY MORPHOLOGIC AND FUNCTIONAL ALTERATIONS.THE MOST SIGNIFICANT OF WHICH ARE DEDIFFERENTIATION OR ANAPLASIA. STRUCTURAL ANAPLASIA IS ACCOMPANIED WITH FUNCTIONAL ANAPLASIA OF QUANTATIVE / QUALITATIVE NATURE. ALL MALIGNANT ANAPLASIAS [EXCEPT GLIOMAS & BASAL CELL CARINOMAS] METASTASISE. BOTH BENIGN AND MALIGNANT NEOPLASM CAUSE LOCAL EFFECTS ON THE HOST DUETO THEIR SIZE AND LOCATION. COMPRESSION, OBSTRUCTION, INFILTRATION, ULCERATION, TISSUE DESTRUCTION AND HAEMORRHAGE ARE THE VITAL OUTCOMES OF THE LOCAL EFFECTS. PATIENTS WITH ADVANCE AND DISSEMINATED CANCERS TERMINALLY HAVE ASTHENIA [EMACIATION], AND ANOREXIA, MALABSORPTION, ULCERATION, NECROSIS, HAEMORRHAGE, INFECTION, PAIN, HYPERMETABOLISM, PYREXIA, ANXIETY AND INSOMNIA. MISCELLANEOUS EFFECTS OF CANER ARE HAEMATOLOGIC CHANGES [ANEMIA, LEUKAEMOID REACTION, DISSEMINATED INTRAVASCULAR COAGULATION AND HYPERCOAGULABILITY], VASCULAR CHANGES [THROMBOPHLEBITIS MIGRANS AND NONBACTERIAL THROMBOTIC ENDOCARDITIS], OSTEOPOROSIS, OSTEOMALACIA, THE CLUBBING OF FINGERS SPECIALLY IN THE CANER OF THE LUNG, AND OTHER KNOWN NEOPLASTIC SYNDROME. INVASIVE CANCER AFFECTS ONE IN THREE OF THE POPULATION AND ONE IN FIVE DIE FROM CANCER.

SURGERY MAY BE ONLY TREATMENT FOR MOST PATIENTS WITH A TUMOUR / MALIGNANT GROWTH. ANOTHER OPTION IS THE USE OF IONIZING RADIATION TO KILL CANCER CELLS. ABOVE ALL, ALMOST EVERY CANCER PATIENT NEEDS CHEMOTHERAPY TO GET CONSOLING MEDICAL TREATMENT EFFECTS. THERE ARE MANY CHEMOTHERAPEUTIC AGENTS AVAILABLE FOR CANCER TREATMENT. SOME MAJOR CLASSES OF  THEM ARE ALKYLATING AGENTS, ANTIMETABOLITES, VINCA ALKALOID,ANTIBIOTICS, INTERFERONS, HORMONS, IMATINIB, CYTARABINE, ARSENIC TRIOXIDE AND IMMUNOTHERAPY. ABOVE ALL, STEM CELLS TRANSPLANTATION FOR SOME TYPES OF BLOOD CANCER IS UNDER ACTIVE ATTEMPTS.

CHEMOTHERAPEUTIC TREATMENT OF NEWLY DIAGNOSED PATIENTS IS USUALLY DIVIDED INTO TWO PHASES: INDUCTION & POSTREMISSION MANAGEMENTS. ONCE RELAPSE OCCURES AFTER THE STANDARD ‘INDUCTION – POSTREMISSION’ CHEMOTHERAPY PATIENTS ARE RARELY CURED. INFECTIOUS COMPLICATIONS REMAIN THE MAJOR CAUSE OF MORBIDITY AND DEATH EVEN DURING ‘INDUCTION – POSTREMISSION’ CHEMOTHERAPY.

‘MASSIVE LYSIS / NECROSIS OF METASTASES AND BLAST CELLS–CRISIS’ STAND GREAT CHALLENGE TO TREATING PHYSICIANS AND CURSE FOR THE CANCER PATIENTS. TO INCREASE THE CURE RATE OF THE MALIGNANT ANAPLASTIC CRISIS / EMERGENCY THE BENEFIT OF MODERN INTENSIVE THERAPY HAS PROVEN HOPELESS. THEREFORE PERSUIT OF NOVEL THERAPIES AS CONSOLIDATION FOR THESE PATIENTS HAS RECENTLY BEEN ACTIVELY PURSUED. NOVEL CANCER TREATMENT OPTIONS ARE APPRECIATED WITH AN ATTENTION FOCUSED ON THEIR OUT COMES, RISKS, AND TOXICITIES.

ACQUIRED DRUG RESISTANCE OF TUMOUR CELLS DUE TO THEIR HIGH INTRINGIC MUTATION RATE IS A MAJOR CAUSE OF TREATMENT FAILURE IN HUMAN CANCERS. THE CLINICAL OUTCOMES OF PATIENTS WITH BLOOD CANCERS TREATED WITH IMATINIB, CYTARABINE, AND OTHER AGENTS / DRUGS ARE VARIABLE, RATHER, UNSATISFACRORY. ALLOGENEIC ‘STEM CELLS TRANSPLANTATION’ IS ONLY TREATMENT OF CHOICE FOR SOME KINDS OF BLOOD CANCERS. HOWEVER, IT IS COMPLICATED BY A HIGH EARLY MORTALITY RATE OWING TO THE TRANSPLANT PROCEDURE.

INTERFERON THERAPY COMBINED WITH OTHER THERAPEUTIC AGENTS AND PROCEDURES HAS BEEN EXCLAIMED IN GETTING SOME RELIEF TO CANCER PATIENTS.

INTERFERONS ARE A COMPLEX GROUP OF NATURALLY OCCURING PROTEINS PRODUCED BY EUKARYOTIC CELLS IN RESPONSE TO VIRUSES, ANTIGENS, AND MITOGENS. THREE DISTINCT GROUPS OF INTERFERONS’ SPECIES HAVE BEEN IDENTIFIED: IFN-ALPHA, IFN- BETA, AND IFN- GAMMA. ALTHOUGH VARIOUS INTERFERONS HAVE BECOME AVAILABLE FOR CLINICAL INVESTIGATION, MOST DATA HAVE BEEN GENERATED WITH IFN – ALPHA PREPARATIONS.

LIKE INTERFERONS AUTOPATHY AAA HAVE POTENT PLEIOTROPIC BIOLOGIC EFFECTS, SPANNING A SPECTRUM OF ANTIVIRAL, MICROBICIDAL, IMMUNOMODULATORY, ANTIPROLIFERATIVE [ANTINEOPLASTIC / ANTIANAPLASTIC], AND ANTIPATHOPHYSIOLOGIC.WHILE IT DOWNREGULATE THE EXPRESSION OF SEVERAL ONCOGENES AND CYTOKINES, THEY ALSO UPREGULATE THE EXPRESSION OF THEIR REGULATOTY FACTORS, ADHESION OF MOLECULES, AND HISTOCOMPATIBILITY GENES.THEY [AAA] ALSO INHIBIT ANGIOGENESIS, INDUCE A CELLULAR & ANTIBODIES RESPONSES, AND ENHANCES APOPTOTIC  ACTIVITIES.

IN CONTRARY TO INTERFERONS, AAA ARE ALPHA, BETA AND GAMMA AUTO IMMUNOGLOBULIN PEPTIDES OF VARIOUS NATURE DEPENDING ON THE INFLICTED DISEASES, AND PREPARED [IN GENERAL] FROM AUTO BLOOD OF THE PATIENTS ITSELF. IT DONOT DEVELOP ANY ACUTE OR CHRONIC SIDE EFFECTS. ACUTE SIDE EFFECTS [FLULIKE SYMPTOMS] AND TACHYPHYLAXIS NEVER APPEAR. CHRONIC REACTIONS SUCH AS FATIGUE AND LETHARGY, DEPRESSION, WEIGHTLOSS, MYALGIAS, AND ARTHERITIS, COUGH, POSTNASAL DRIP, DRYNESS OF SKIN, IMMUNE MEDIATED THROMBOCYTOPENIA, ANEMIA, AND ANY OTHER AUTOIMMUNE SIDE EFFECTS [HYPOTHYROIDISM], ARE NEVER SEEN.

CANCER PATIENTS TREATED WITH AAA – THERAPY ALONE OR INCOMBINATION OF THE ‘TREAMENT – ASSIST’ NEVER DEVELOP ANY NEUROLOGIC TOXICITY [SAY, LETHARGY] OR IMPOTENCY, LOSS OF SEXUAL LIBIDO, OR ANY CARDIOVASCULAR, OPHTHALMIC [etc] COMPLAIN.

DETAIL PATHOGENESIS OF NEOPLASIA / ANAPLASIA / CANCER ON MOLECULAR / GENETICAL [ENERGY / MATERIAL EXCHANGE] LEVEL PERCIEVED BY AUTOPATHY MEDICAL SCIENCE HAS BEEN DESCRIBED BELOW [SEE, THE ENERGY TRIANGALS].

THE GREAT CHALLENGE BEING FACED BY PHYSICIANS IN TREATING PATIENTS WITH MASSIVE LYSIS OF ‘WIDE SPREAD METASTASES, AND BLAST CELL – CRISIS REDUCES TO AN AMICABLE LEVEL IF AUTOPATHY APPROACH OF TREATMENT IS COMBINED WITH STANDARD ALLOPATHIC APPROACHES [TREATMENT – ASSIST]. SUCH PROSPECTIVE PATIENTS ARE INFUSED [INTRAVENOUS] APPROPRIATE QUANTITY [500 TO 1000 microgram] OF BETA FRACTION OF AAA PREPARED EITHER FROM THE PATIENTS OWN BLOOD OR FROM ANY HISTOCOMPATIBLE ALLOGENEIC BLOOD. MOST OF THE PATIENTS EITHER DONOT DEVELOP ‘CRISIS’ OR THE GRADE OF THE CRISIS BECOMES MANAGABLE.THE DRUGS, WHICH GENERALLY HAD BECOME RESISTANT, AND OF NO USE, TURN HIGHLY EFFECTIVE AND ITS COURSE OF TREATMENT BECOMES SMALLER & ECONOMIC. IT IS ESSENTIAL TO NOTE THAT THE BETA – FRACTION OF THE AAA, SO PREPARED IS PROPERLY WASHED WITH ETHER BEFORE ITS INFUSION WITH THE ‘TREATMENT – ASSIST’. ALL PATIENTS WITH CANCERS ARE PRESCRIBED AAA – AUTOPATHY TREATMENT TO GET COMPLETE REMISSION / CURE.ITS DOSES AND REGIMES ARE [250 TO 750 microgram] CALCULATED BASED ON THE SEVERITY OF THE DISEASE, AUTOPATHY TEST REPORTS, AND OTHER ALLOPATHIC PARAMEDICAL FINDINGS. PATIENTS WHO ARE TO BE GIVEN AAA – AUTOPATHY TREATMENT ALONG WITH THE ‘TREATMENT – ASSIST’ ARE STRICTLY AVOIDED BLOOD TRANSFUSION. POSTREMISSION TREATMENTS ARE OFTEN NOT REQUIRED. ONCE A YEAR MASTER – CHECK UP IS DONE, AND AAA- AUTOPATHY TREATMENT IS REPEATED AT LEAST FOR THREE TO FIVE CONSEQUENT YEARS. DURING POSTREMISSION PERIOD MANY CANCER PATIENTS SHOW COMPLAINLESS LIVING IF THEY ARE TAKING 150 ml LEMON – HOT WATER DRINK TDS, CHROMIUM CITRATE 300 / 500 microgram OD, HOMEOPATHY PREPARATION ‘ARSENICUM – Q’ A DROP SUBLINGUALLY EVERY FORTNIGHT, ALON WITH ANTIOXIDANTS / VITAMINS AND SYMPTOMATIC [IF NEEDED] ALLOPATHY TREATMENT.

PATHOGENESIS OF THE MALIGNANT CELL DIVISION IN THE LIGHT OF ‘THE NATURAL LAWS OF THE VIRTUAL ENERGY'  EXERCISING ON CELLULAR LEVEL IN THE LIVING IDENTITIES.

THE NATURAL LAWS OF THE VIRTUAL ENERGY KEEP ON EXERCISING ALL OVER THE UNIVERSE IN NON-LIVINGS AND LIVINGS BOTH. IN NON-LIVINGS THIS ENERGY EXPRESSION HAS BEEN BROADLY DEFINED IN ‘BIHARI-SIR’ SCIENCE & TECHNOLOGY, WHICH IS SIMILARLY REPRESENTED IN TRIANGLES AS DEPICTED ABOVE.

            IN THE LIVING IDENTITIES NUCLEAR DIVISION IS ITS BURNING EXAMPLE. IN THIS PROCESS OF A CELL DIVISION (SEE ABOVE NOTED ENERGY TRIANGLES) NPS & SECOND MESSENGER PLAY AS MOTIVE FORCE IN ASSISTANCE WITH CENTRIOLE AND MITOCHONDRIAL ACTIVATION. THE ACTIVITIES OF THE NUCLEUS PER SE REPRESENT EXPRESSION OF THE VIRTUAL ENERGY.

                      TREATMENT OF CANCER LOOKS QUITE POSSIBLE IF A CURATIVE APPROACH IS ALIGNED WITH THE EXPRESSION OF THE NATURAL LAWS OF THE VIRTUAL ENERGY MECHANISM AS PERCEIVED ABOVE WITH THE CELL DIVISIONS.

                      ACCORDING TO THE AUTOPATHY DICTUM OF THOUGHTS DISEASES CAN BE ATTACKED BY COMMANDING ON THE ACTIVITIES OF CENTROSOMAL & CHROMOSOMAL HOUSINGS OF THE CYTOLOGICAL CONTROL BEING GOVERNED BY NUCLEOLUS.

                    NOW IT IS CLEAR TO US THAT THE NUCLEUS IS THE SITE FOR VIRTUAL ENERGY EXPRESSION. THE NUCLEOLUS IS THE MOTIVE FORCE GOVERNING ELEMENT INDUCING THE PROCESS OF THE CELL DIVISION.           

      THE HUMAN BODY IS MADE UP OF CELLS AND OF INTRACELLULAR SUBSTANCES PROVIDED BY THE CELLS. MANY CELLS OF THE BODY HAVE A LIMITED SPAN OF FUNCTIONAL ACTIVITIES AT THE END OF WHICH THEY UNDERGO DIVISION INTO TWO DAUGHTER CELLS. THE PERIOD BETWEEN TWO SUCCESSIVE CELL DIVISIONS IS CALLED INTERPHASE. DURING THIS PERIOD MATURATION OF THE CELLS TAKES PLACE. A MATURED CELL GETS ARRANGED ALL THE REQUIREMENTS FOR ITS NUCLEAR PROLIFERATION OR CELL DIVISION. DURING CELL DIVISION TREMENDOUS QUANTUM OF BIO-ENERGY IS NEEDED WHICH IS PROVIDED BY HIGHLY ACTIVATED MATERNAL MITOCHONDRIAS. DURING CELL DIVISION NUMEROUS CHANGES OCCUR IN THE CHROMOSOMES. WHILE THESE CHANGES ARE OCCURRING A NUMBER OF OTHER EVENTS ALSO TAKES PLACE INSIDE THE CELL. MATURED NORMAL CELL, SURROUNDED BY ADDITIONAL REPLICATING MATERIALS FILLED IN INTRACELLULAR SPACES, IS SUBJECTED TO STRONG EXTRACELLULAR ‘NUCLEAR PROLIFERATIVE STIMULI’ (NPS) THESE STIMULI PROVOCATE SECOND MESSENGER INSIDE THE ‘EXTRANUCLEAR AREA’ OF THE CELL. THE SECOND MESSENGER ACTIVATES NUCLEOLUS RESIDING INSIDE THE ‘INTRANUCLEAR AREA’ TO ASSESS THE PREPAREDNESS OF THE CELL WITH REPLICATIVE STRUCTURAL MATERIALS. HAVING FOUND ITS SATISFACTORY QUANTUM THE NUCLEOLUS GIVES COMMAND TO THE CENTROSOMAL SYSTEM AND TO THE MITOCHONDRIAL ARRANGEMENTS TO GO AHEAD WITH THE CELL DIVISION PROCESS. AND THE NUCLEOLUS DISAPPEARS FROM THE SITE. THE SECOND MESSENGER ENGINEERS WHOLE PROCESS OF THE CELL DIVISION USING EXTRACELLULAR, INTRACELLULAR AND INTRANUCLEAR RESOURCES ON EXPENSE OF THE MITOCHONDRIAL ENERGY DISTRIBUTED AS SHOWN IN THE BIO-ENERGY TRIANGLES –1. THE EFFECTS OF THIS ENERGY EXPRESSION MAY BE SUMMARIZED AS UNDER.            

THE TWO CENTRIOLES SEPARATE AND MOVE TO OPPOSITE POLES OF THE CELL. THE NUCLEAR MEMBRANE BREAKS DOWN AND NUCLEOLUS DISAPPEARS. AT THE END OF THE CELL DIVISION THE NUCLEOLUS REAPPEARS. THE CENTRIOLE IS NOW DUPLICATED AT THIS STAGE OR IN EARLY INTERPHASE. IN THIS PROCESS THE ORGANELLS ARE ALSO DUPLICATED AND EACH DAUGHTER CELL COMES TO HAVE FULL COMPLEMENT OF THEM.THE INTERPHASE PROCEEDS AGAIN FOR THE CELL MATURATION.       

MANY HUMAN NEOPLASMAS ARE ASSOCIATED WITH NONRANDOM CHROMOSOMAL ABNORMALTIES, SUGGESTING THAT CERTAIN CYTOGENIC ABNORMALTIES MUST BE IMPORTANT AND PRIMARY EVENT IN NEOPLASTIC TRANSFORMATION. BASED ON LITERATURE, OWN EXPERIMENTAL AND CLINICAL EXPERIENCE IT HAS BEEN CONCEIVED THAT PATERNAL MITOCHONDRIAS PRESENT IN SIDE THE EXTRANUCLEAR SPACE OF A CELL ARE SUBJECTED TO GENE AMPLIFICATION. THESE MUTANT MITOCHONDRIAS PLAY AS PRIMARY/ BASIC EVENT IN NEOPLASTIC TRANSFORMATION.

‘NUCLEAR PROLIFERATIVE STRONG STIMULI’ (NPS) ON THE CELL MEMBRANE OF A PROSPECTIVE CELL ARE PROTESTED AT ITS PRIMARY SITE IN TWO STAGES: BY DISPLASIA AND ANAPLASIA. THE FIRST SUCH MANIFESTATION IS PREDECESSOR OF THE SECOND. IF THE STIMULI PROVOCATING MATERIALS ARE NOT ELIMINATED BY DISPLASIA THE PROCESS OF ANAPLASIA DEVELOPS AS ULTIMATE TOOL OF DEFENSE. THIS IS EXPRESSED AS GENE AMPLIFICATION IN THE PATERNAL MITOCHONDRIAS (MMS), AND PLEOMORPHISM CAUSED BY THE SECOND MESSENGER (SMS). IN THE NEXT GENERATION OF THE ANAPLASTIC CELLS THE DUTIES OF THE NUCLEAR PROLIFERATIVE STIMULI & SECOND MESSENGER BOTH ARE PERFORMED BY THESE PATERNAL MUTANT MITOCHONDRIAS (MMS); WHICH TOO GET REPLICATED AND TRANSFERRED TO THE DAUGHTER CELLS CAUSING GENERATION OF ADDITIONAL CENTRIOLE (ACL) SPINDLES WITHOUT DISAPPEARANCE OF THE NUCLEOLI (NLA). THEY TOO LOOK PROMINENT IN THE PLEOMORPHIC NEOPLASTIC CELLS.

THE SURROUNDING DISPLASTIC CELLS AT THE PRIMARY SITE OF EVENTS KEEP ON MANUFACTURING & SUPPLYING REPLICATION STRUCTURAL MATERIALS FOR UNINTERRUPTED NEOPLASTIC CELL DIVISION. OTHER BASIC FEATURES OF THE NEOPLASTIC CELL DIVISION SCARCELY DIFFER FROM NORMAL CELL DIVISION EXCEPT THOSE GENETICAL TRANSFORMATIONAL OUT-PUTS RELATED TO ACTIVATED BALANCED TRANSLOCATION, DELETION OF CHROMOSOMES AND MANY OTHER GENES AMPLIFICATION. THIS EVENT OF MUTANT PATERNAL MITOCHONDRIAS’  (MMS) REPLICATION IS EXPRESSED AS LOCAL IGNORANCE OF THE DEFENCE MECHANISM IN CASE OF CANCER/NEOPLASTIC GROWTH. WHEREAS, SUCH MUTATION IN MATERNAL MITOCHONDRIAS (MMM) TO EXPRESSED AS GENERALISED IGNORANCE OF THE DEFENCE MECHANISM, AS IT HAPPENS IN CASE OF AIDS. THIS BIO-ENERGY ACTION MECHANISM IN NEOPLASTIC CELL DIVISION HAS BEEN SHOWN IN THE BIO-ENERGY TRIANGLES –2.

HAVING IN MIND THE ABOVE NOTED MECHANISM OF THE NEOPLASTIC CELL DIVISION THERE SHOULD BE FOLLOWING MEDICAL APPROACHES OF THE CANCER TREATMENT:  SURGICAL, CHEMOTHERAPY, RADIATION THERAPY, BIOLOGICAL MODIFIER’S THERAPY AND AUTOIMMUNE (AUTOPATHY) THERAPY.

    AUTOPATHY THERAPY AS DESCRIBED ABOVE, SHOULD INVOLVE ANTIOXIDANT THERAPY TO COMBATE OXIDATIVE PHOSPHORYLATION PROCESS INDUCED BY PATERNAL MUTANT MITOCHONDRIAL (MMS) ACTIVATION. AUTOIMMUNOTHERAPY PROPOSED BY THE AUTOPATHY MODE OF TREATMENT SHOULD ATTACK ON BOTH SITES OF THE NEOPLASTIC CELLS TO KILL MUTANT MITOCHONDRIAS AND THE ACTIVATED / TRANSFORMED NUCLEOLI (NLA), i.e. EXTRA & INTRA NUCLEAR IMMUNOMODULATORY EFFECTS. EXTRA NUCLEAR IMMUNOTHERAPY MAY BE PROVIDED DIRECTLY WITH THE HELP OF SPECIFIC IMMUNE PEPTIDES, i.e. AAA-I, AND THE INTRANUCLEAR IMMUNOTHERAPY SHOULD BE PROVIDED USING SPECIFIC IMMUNE PEPTIDES BINDED WITH STEROIDS, i.e. AAA-II.  

  A CLINICIST DEALING WITH TREATMENT OF MALIGNANCY SHOULD PROVIDE NOT ONLY ABOVE NOTED AUTOPATHY ANTI-ANTIBODIES THERAPY [AAA THERAPY], BUT IT MUST BE ASSOCIATED WITH OTHER IMMUNODEPRESANTS TO COUNTER NUMEROUS CHAINS OF IMMUNE REACTIONS. AMONG MANY SUCH DRUGS ENDOXAN, IMMURAN, CORTICOSTEROIDS, HYDREA AND MANY SUCH OTHERS COULD SHOW DESIRABLE OUTCOMES. TO MAINTAIN HYDRATION INSIDE THE NEOPLASTIC CELLS PROGESTRON AND CORTICOSTEROIDS (ORGAMED, FARLUTAN, DEXONA etc.) MAY BE USED. TO PROVIDE ANTIOXYDANTS AND ANTIMETABOLITES TO COUNTER MITOCHONDRIAL AND NUCLEAR ACTIVATION – METHOTREXATE ALONG WITH FREECAD, GLYNASE, PERITOL etc MAY BE ADMINISTERED SUCCESSFULLY. FOR DETOXIFICATION & INFLAMATORY PROCESSES BROAD SPECTRUM ANTIBIOTICS, CORTICOSTEROIDS, DELORTAN / ANTI HISTAMINES, INTRAVENOUS FLUID (BLOOD) TRANSFUSION TREATMENT MAY YIELD SATISFACTORY OUT PUTS.

   AMONG A NUMBER OF CANCER PATIENTS ABOVE NOTED MIXED THERAPEUTIC APPROACHES HAVE PROVED TO BE VERY MUCH EFFECTIVE.

ABBREVIATIONS USED IN THE BIO-ENERGY TRIANGLES ARE AS UNDERSTOOD – NPS = NUCLEAR PROLIFERATIVE STRONG STIMULI; MRA = MATERNAL MITOCHONDRIAL ACTIVATION; CSA = CENTROSOMAL ACTIVATION; SMA = SECOND MESSENGER ACTIVATION; CLA = CENTRIOLES’ ACTIVATION; NPF = NUCLEAR PROLIFERATION; MMS = PATERNAL MUTANT MITOCHONDRIAL STRONG STIMULI AND THE FUNCTION OF THE SECOND MESSENGER DEVELOPED BY IT; ACL = ADDED CENTRIOLES’ ACTIVATION; AND NLA = NUCLEOLI ACTIVATION. THE ARROWS SHOW THE DIRECTION OF BIO-ENERGY ACTIONS / MOVEMENTS.